全球13个肥胖儿中就有中国娃
2009-5-18 17:35:31 出处：北京晚报
5月20日是中国学生营养日。中国学生营养与健康促进会发布的一份报告指出，我国有1200万超重肥胖的儿童少年，全世界1.55亿超重肥胖儿童少年中，每13个里就有一个是中国儿童少年。不吃早餐、经常吃西式快餐和身体活动减少是我国儿童发生肥胖的主要危险因素。
此前市卫生局发布的健康播报显示，近年来，北京市儿童青少年肥胖呈明显上升趋势，目前2至18岁儿童青少年肥胖率接近10%，还有11%的同龄儿童处于超重状态，极可能发展为肥胖。
昨天发布的报告指出，不健康的饮食行为是引起儿童少年肥胖的一个主要原因。调查显示，不吃早餐的儿童少年发生肥胖的危险是每天食用早餐儿童的1.7倍。不吃早餐的儿童少年容易发生肥胖的原因，可能是到吃午餐时饥不择食，不知不觉吃下去过多的食物而引起能量摄入过多，造成体内脂肪蓄积。
经常吃西式快餐被认为可能是肥胖发生的重要因素之一。调查分析发现，我国城市儿童少年食用西式快餐频率每月大约1次者，发生肥胖的危险是不吃快餐者的1.3倍。
另外，经常喝含糖饮料也是引起体重增加的重要原因。儿童少年喝营养素含量低、能量高的碳酸饮料越多，他们肥胖的可能性就越大。同时因较少饮用牛奶和其他有营养饮品，使蛋白质、钙、磷、镁和维生素A等营养素的摄入量减少。
除了膳食因素，身体活动不足也是导致儿童少年肥胖增多的重要原因。调查显示，我国6至12岁儿童经常参加锻炼的比例仅为4.7%；13至17岁的儿童少年经常参加锻炼的比例仅为8.1%。
中国学生营养与健康促进会已在北京启动了“带着水果去旅行”的大型公益活动，鼓励小学生每天合理摄食一至两份水果，培养均衡营养膳食的观念和健康科学的生活方式。
2010年1月15日星期五
2010年1月7日星期四
Yolanda 享受食物, 不用擔心任何反彈
姓名: Yolanda Chiu
職業: 資深輔導員
現况: 進入終身維持期
減重只用了一個週期
纖體前 112磅 纖體後 104磅
纖體前 28% 纖體後 17%
參加「健康行」的原因分享:
最初進展緩慢, 教練發現我的新陳代謝極慢, 我選擇信任我的教練並且跟隨指導,現在已進入終身維持期, 我享受食物, 不用擔心任何反彈, 而營養保充品使我身體得到調理, 皮膚有彈性及光澤, 精神狀態改善而且精力旺盛 。
2010年1月6日星期三
夢家班培訓中心開幕典禮 暨 HP PRO Team 啟動儀式
各界嘉賓雲集
大會司儀Ronald Chan(Hillman大公子)
HP PRO TEAM 工作室及網頁正式啟動
特別嘉賓 - 蘭薇老師(專家顧問團隊之一)
表演嘉賓 - Dick Wong
Mental Magician - Mr. Romeo
2010年1月5日星期二
U.S. agencies want to ban some kid food ads
U.S. agencies want to ban some kid food ads
WASHINGTON
Tue Dec 15, 2009 5:39pm EST
WASHINGTON (Reuters) - In a bid to tackle rising youth obesity, U.S. companies would be prohibited from advertising to children foods that contain large amounts of sugar or salt, or even low levels of trans fats, under a proposal released on Tuesday by a working group from several U.S. agencies.
BARACK OBAMA | HEALTH | LIFESTYLE
The working group made up of members of the Food and Drug Administration, Federal Trade Commission, U.S. Department of Agriculture and Centers for Disease Control issued what it called tentative proposed standards for food marketed to children, defined as up to age 17.
Those foods could not have more than 1 gram of saturated fat per serving, 13 grams of added sugar, 200 milligrams of sodium or 0 trans fats, which they defined as more than half a gram, per normal serving.
At a related conference to discuss food advertising and any link it might have to obesity among children, Kathleen Sebelius, secretary of Health and Human Services, said that if the obesity-related health risks -- high blood pressure and diabetes among them -- were caused by radiation "alarm bells would be going off across America. There would be a huge outcry."
Sebelius, who also admitted to a weakness for Cheetos, said that it was important for any changes in advertising rules to be across the board so that companies that eliminate child-oriented ads for unhealthy foods were not punished for it.
"We need to start doing a better job of regulating the types of ads our children see," she added.
Some food manufacturers have already reformulated some kid favorites to take health concerns into account. Kellogg, which makes Froot Loops, and General Mills, maker of Cocoa Puffs, have both said they would reduce the amount of sugar in some food advertised to children.
The chairman of the FTC, Jon Leibowitz, noted these and other steps forward.
"These changes have come in small increments," said Leibowitz. "Put simply, it is time for industry to supersize its efforts."
Dan Jaffe, executive vice president for government relations at the Association of National Advertisers, argued that advertisers were not to blame for the growing number of fat children and any restrictions on ads could run afoul of the First Amendment.
"The advertising community faces a real clear and growing threat of censorship," he said.
(Reporting by Diane Bartz, editing by Matthew Lewis)
"http://www.reuters.com/article/idUSTRE5BE5BO20091215?feedType=RSS&feedName=healthNews&utm_source=feedburner&utm_medium=feed&utm_campaign=Feed:+reuters/healthNews+(News+/+US+/+Health+News)&utm_content=Bloglines"
WASHINGTON
Tue Dec 15, 2009 5:39pm EST
WASHINGTON (Reuters) - In a bid to tackle rising youth obesity, U.S. companies would be prohibited from advertising to children foods that contain large amounts of sugar or salt, or even low levels of trans fats, under a proposal released on Tuesday by a working group from several U.S. agencies.
BARACK OBAMA | HEALTH | LIFESTYLE
The working group made up of members of the Food and Drug Administration, Federal Trade Commission, U.S. Department of Agriculture and Centers for Disease Control issued what it called tentative proposed standards for food marketed to children, defined as up to age 17.
Those foods could not have more than 1 gram of saturated fat per serving, 13 grams of added sugar, 200 milligrams of sodium or 0 trans fats, which they defined as more than half a gram, per normal serving.
At a related conference to discuss food advertising and any link it might have to obesity among children, Kathleen Sebelius, secretary of Health and Human Services, said that if the obesity-related health risks -- high blood pressure and diabetes among them -- were caused by radiation "alarm bells would be going off across America. There would be a huge outcry."
Sebelius, who also admitted to a weakness for Cheetos, said that it was important for any changes in advertising rules to be across the board so that companies that eliminate child-oriented ads for unhealthy foods were not punished for it.
"We need to start doing a better job of regulating the types of ads our children see," she added.
Some food manufacturers have already reformulated some kid favorites to take health concerns into account. Kellogg, which makes Froot Loops, and General Mills, maker of Cocoa Puffs, have both said they would reduce the amount of sugar in some food advertised to children.
The chairman of the FTC, Jon Leibowitz, noted these and other steps forward.
"These changes have come in small increments," said Leibowitz. "Put simply, it is time for industry to supersize its efforts."
Dan Jaffe, executive vice president for government relations at the Association of National Advertisers, argued that advertisers were not to blame for the growing number of fat children and any restrictions on ads could run afoul of the First Amendment.
"The advertising community faces a real clear and growing threat of censorship," he said.
(Reporting by Diane Bartz, editing by Matthew Lewis)
"http://www.reuters.com/article/idUSTRE5BE5BO20091215?feedType=RSS&feedName=healthNews&utm_source=feedburner&utm_medium=feed&utm_campaign=Feed:+reuters/healthNews+(News+/+US+/+Health+News)&utm_content=Bloglines"
2010年1月4日星期一
Adverse consequences of obesity may be greater than previously thought
Adverse consequences of obesity may be greater than previously thought
Public release date: 22-Dec-2009
Contact: Emma Dickinson
edickinson@bmj.com
44-207-383-6529
BMJ-British Medical Journal
Research: The association between body mass index and mortality using offspring BMI as an indicator of own BMI: Large intergenerational mortality study
The link between obesity and cardiovascular mortality may be substantially underestimated, while some of the adverse consequences of being underweight may be overstated, concludes a study published on bmj.com today.
This means that the adverse influence of higher BMI and obesity in a population is of greater magnitude than previously thought, say the authors.
Numerous studies have already investigated the link between body mass index (BMI) and mortality. They show that high BMI is associated with higher rates of death from cardiovascular causes, diabetes, and some cancers, while low BMI is associated with increased mortality from other causes, such as respiratory disease and lung cancer.
But there are inconsistencies in the evidence that low body mass index actually increases the risk of causes of death such as respiratory disease and lung cancer.
Some researchers argue that this association may be biased by a process called reverse causality, where a severe illness, such as lung cancer, leads to both weight loss and higher mortality. Other factors such as smoking and poor socioeconomic circumstances may also lead to biasing estimates. This is known as confounding.
So a team from the University of Bristol and the Karolinska Institute in Sweden set out to obtain a valid estimate of the association between body mass index (BMI) and mortality by comparing, for over one million Swedish parent-son pairs, the BMI of the sons as young adults with mortality among their parents.
Using offspring BMI as an indicator of parental BMI avoids problems of reverse causality and is less influenced by confounding, explain the authors.
Their analysis shows strong associations between high offspring BMI (used as a so-called instrumental variable) and parental mortality from cardiovascular disease, diabetes, and some cancers, as reported in other studies of own BMI with mortality. However, unlike previous studies, there was no evidence of an association between low BMI and an increased risk of respiratory disease and lung cancer mortality.
These findings suggest that the apparent adverse consequences of low BMI on respiratory disease and lung cancer mortality may be overstated, whereas the adverse consequences of higher BMI on cardiovascular disease mortality may be substantially underestimated, say the authors.
These conclusions have important implications for public health practice because they suggest that reducing population levels of overweight and obesity (or preventing their rise) will have a considerable benefit to population health, they add. Suggestions to the contrary, which have received considerable media attention over recent years, are probably misguided.
Public release date: 22-Dec-2009
Contact: Emma Dickinson
edickinson@bmj.com
44-207-383-6529
BMJ-British Medical Journal
Research: The association between body mass index and mortality using offspring BMI as an indicator of own BMI: Large intergenerational mortality study
The link between obesity and cardiovascular mortality may be substantially underestimated, while some of the adverse consequences of being underweight may be overstated, concludes a study published on bmj.com today.
This means that the adverse influence of higher BMI and obesity in a population is of greater magnitude than previously thought, say the authors.
Numerous studies have already investigated the link between body mass index (BMI) and mortality. They show that high BMI is associated with higher rates of death from cardiovascular causes, diabetes, and some cancers, while low BMI is associated with increased mortality from other causes, such as respiratory disease and lung cancer.
But there are inconsistencies in the evidence that low body mass index actually increases the risk of causes of death such as respiratory disease and lung cancer.
Some researchers argue that this association may be biased by a process called reverse causality, where a severe illness, such as lung cancer, leads to both weight loss and higher mortality. Other factors such as smoking and poor socioeconomic circumstances may also lead to biasing estimates. This is known as confounding.
So a team from the University of Bristol and the Karolinska Institute in Sweden set out to obtain a valid estimate of the association between body mass index (BMI) and mortality by comparing, for over one million Swedish parent-son pairs, the BMI of the sons as young adults with mortality among their parents.
Using offspring BMI as an indicator of parental BMI avoids problems of reverse causality and is less influenced by confounding, explain the authors.
Their analysis shows strong associations between high offspring BMI (used as a so-called instrumental variable) and parental mortality from cardiovascular disease, diabetes, and some cancers, as reported in other studies of own BMI with mortality. However, unlike previous studies, there was no evidence of an association between low BMI and an increased risk of respiratory disease and lung cancer mortality.
These findings suggest that the apparent adverse consequences of low BMI on respiratory disease and lung cancer mortality may be overstated, whereas the adverse consequences of higher BMI on cardiovascular disease mortality may be substantially underestimated, say the authors.
These conclusions have important implications for public health practice because they suggest that reducing population levels of overweight and obesity (or preventing their rise) will have a considerable benefit to population health, they add. Suggestions to the contrary, which have received considerable media attention over recent years, are probably misguided.
2010年1月3日星期日
Change your eating behavior for 2010
The Allure of “Fast Food”
The Better Life Experts | June 5, 2009
We all know that eating a diet comprised primarily of take-out, fast food, is dangerous to our health. Yet, we continue to feed our bodies with high fat, high calorie and low nutrition foods from drive through restaurants and convenience stores as if we didn’t know any better. Why do we persist in refueling our bodies with nutritionally low-grade food products?
Perception has quite a lot to do with choosing fast food as fuel. We think that it is more convenient to pick something up for lunch or on the way home from work that is already prepared. We believe that it is cheaper than making a lunch or dinner from non-processed foods – Who has the time to cook? Add the general dislike of mainstream American culture for cooking and you have the ingredients that make fast food a growth industry.
Habitual behaviors may also play a big role how we make food choices. Eating fatty foods can be viewed as one of many habits that arise as a result of specific neural activity patterns made in the brain. These familiar neuronal pathways make it difficult for people to break habitual patterns of behavior. Many of us are conditioned during childhood with sweet or fatty foods as rewards or treats. The Easter Bunny brings chocolate, we celebrate our birthdays with cake and ice cream, the Christmas season sends us over the edge with pies, cookies, towers of candy, stuffing, heaps of mashed potatoes, etc. Neural pathways develop which equate feeling good with eating foods that are fatty, sweet and high in calories. These routes through the brain can be viewed as ruts in a road. The more we travel down the same path, the deeper the furrows become. We need to fill in these ruts and build new neural pathways for our brains to follow by breaking poor food habits, which includes substituting healthier choices in place of fast food meals. During the last decade neuroscientists have discovered that our brains are capable of creating an endless number of new neural connections through thought patterning.
Maybe it’s time for some of us to rethink our misconceptions about food and try some new paths on our way to a better life – ones that don’t involve stopping at old haunts on rutted roads.
The Better Life Experts | June 5, 2009
We all know that eating a diet comprised primarily of take-out, fast food, is dangerous to our health. Yet, we continue to feed our bodies with high fat, high calorie and low nutrition foods from drive through restaurants and convenience stores as if we didn’t know any better. Why do we persist in refueling our bodies with nutritionally low-grade food products?
Perception has quite a lot to do with choosing fast food as fuel. We think that it is more convenient to pick something up for lunch or on the way home from work that is already prepared. We believe that it is cheaper than making a lunch or dinner from non-processed foods – Who has the time to cook? Add the general dislike of mainstream American culture for cooking and you have the ingredients that make fast food a growth industry.
Habitual behaviors may also play a big role how we make food choices. Eating fatty foods can be viewed as one of many habits that arise as a result of specific neural activity patterns made in the brain. These familiar neuronal pathways make it difficult for people to break habitual patterns of behavior. Many of us are conditioned during childhood with sweet or fatty foods as rewards or treats. The Easter Bunny brings chocolate, we celebrate our birthdays with cake and ice cream, the Christmas season sends us over the edge with pies, cookies, towers of candy, stuffing, heaps of mashed potatoes, etc. Neural pathways develop which equate feeling good with eating foods that are fatty, sweet and high in calories. These routes through the brain can be viewed as ruts in a road. The more we travel down the same path, the deeper the furrows become. We need to fill in these ruts and build new neural pathways for our brains to follow by breaking poor food habits, which includes substituting healthier choices in place of fast food meals. During the last decade neuroscientists have discovered that our brains are capable of creating an endless number of new neural connections through thought patterning.
Maybe it’s time for some of us to rethink our misconceptions about food and try some new paths on our way to a better life – ones that don’t involve stopping at old haunts on rutted roads.
2010年1月1日星期五
解構各種醣類
解構各種醣類 【明報專訊】
糖是碳水化合物，澱粉質與纖維素也是。箇中的分別，是結構的不同。
碳水化合物（carbohydrates），又稱作醣類或醣質。按其結構不同，分為單醣、雙醣或多醣。
◆單醣（monosaccharides）
單一結構的醣：
(葡) 葡萄糖（glucose或dextrose）
(果) 果糖（fructose）
(半) 半乳糖（galactose）
◆雙醣（disaccharides）
兩個單醣手拖手：
(葡)＋(葡)＝麥芽糖（maltose）
(葡)＋(果)＝蔗糖（sucrose）
(葡)＋(半)＝乳糖（lactose）
◆多醣（polysaccharides）
由3個或以上的單醣組成：
(葡)＋(葡)＋(葡)＋(葡)＋(葡)＋(葡)
＋(葡)＋(葡)＋(葡)＋(葡)＋(葡)＋(葡)
＝澱粉（starch，儲存在植物內），或
＝纖維（fibre，植物細胞壁）
*澱粉進入消化道後，最終被分解為葡萄糖，提供能量；
**纖維縱然進入消化道，但它的結構不會被拆散成單醣。由於它不會被消化，而是直接排出體外，因而有利腸道健康。
明報2009年10月26日 星期一
糖是碳水化合物，澱粉質與纖維素也是。箇中的分別，是結構的不同。
碳水化合物（carbohydrates），又稱作醣類或醣質。按其結構不同，分為單醣、雙醣或多醣。
◆單醣（monosaccharides）
單一結構的醣：
(葡) 葡萄糖（glucose或dextrose）
(果) 果糖（fructose）
(半) 半乳糖（galactose）
◆雙醣（disaccharides）
兩個單醣手拖手：
(葡)＋(葡)＝麥芽糖（maltose）
(葡)＋(果)＝蔗糖（sucrose）
(葡)＋(半)＝乳糖（lactose）
◆多醣（polysaccharides）
由3個或以上的單醣組成：
(葡)＋(葡)＋(葡)＋(葡)＋(葡)＋(葡)
＋(葡)＋(葡)＋(葡)＋(葡)＋(葡)＋(葡)
＝澱粉（starch，儲存在植物內），或
＝纖維（fibre，植物細胞壁）
*澱粉進入消化道後，最終被分解為葡萄糖，提供能量；
**纖維縱然進入消化道，但它的結構不會被拆散成單醣。由於它不會被消化，而是直接排出體外，因而有利腸道健康。
明報2009年10月26日 星期一
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